Ferdausi, NourinAhmed, Sabit2023-01-092023-01-0920222022-07ID 18136029http://hdl.handle.net/10361/17697This thesis is submitted in partial fulfillment of the requirements for the degree of Bachelor of Science in Biotechnology 2022.Catalogued from PDF version of thesis.Includes bibliographical references (pages 38-63).Numerous attempts are being taken globally to comprehend the molecular mechanisms underlying the coronavirus disease 2019 or COVID-19. The majority of deaths among patients might be a direct result of SARS-CoV-2 infection, as many studies suggest a connection between COVID-19-induced oxidative stress and inflammation with multiple organ system damages. To develop treatments for SARS-CoV-2 infected patients, it is currently clinically critical to infer how oxidative stress and molecular inflammatory pathways drive COVID-19 propagation to extreme phenotypes including neurological disorders, cardiac damage, pulmonary dysfunction, etc. This research reviews the molecular pathophysiology of SARS-CoV-2 and how it relates to oxidative stress and inflammation-induced organ damage to the heart, liver, kidney, brain, lungs, and other major organs, as well as the development of cancer. A summary of potential antioxidant treatments that might prevent or minimize the severity of the disease is also discussed.63 pagesenBrac University theses are protected by copyright. They may be viewed from this source for any purpose, but reproduction or distribution in any format is prohibited without written permission.SARS-CoV-2Oxidative stressInflammationMultiple organ damageCancer developmentAntioxidant therapyCOVID-19 (Disease)--DiagnosisThesis